Nonalcoholic fatty liver disease , a condition associated with obesity can range in severity from simple steatosis to nonalcoholic steatohepatitis to cirrhosis of the liver, resulting in death may but http://generic-tadalafil.com . The progression of the disease. The prevailing hypothesis two hits are required. First is overeating which causes the accumulation of fat in the liver, prolonged. ‘s second goal has been hypothesized to oxidative stress, mitochondrial include violations or inflammatory cytokine production by regulatory T cells . A normal diet. Recently a major player a major player in the liver, immune regulation, the researchers, led by Xiong Ma of Johns Hopkins University in Baltimore studied, its role in determining the pathogenesis of NASH They hypothesized that the regulation of Tregs reducing makes a fatty liver, inflammation worse when the organ is exposed to secondary .
To kill, toge and high-fat – diet -linkedA high fat diet may regulatory T cells in the liver to kill, to develop so that steatosis to steatohepatitis , according to the results of a new study in the November issue of Hepatology, a journal published by John Wiley & Sons on behalf of the American Association for the Study of Liver Diseases published. The article is also available online at Wiley Interscience (.
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